Achilles tendon rupture is the most common tendon injury of the lower extremity and has been reported with increasing frequency, particularly in men between 30 and 50 years of age who lead a sedentary lifestyle and suddenly participate in recreational sports. Most ruptures occur 2-6 cm proximal to the calcaneal insertion in a relatively hypo vascular zone, where repetitive mechanical loading and intrinsic tendon degeneration act together to weaken the structure. Age-related collagen changes, chronic overuse with microtrauma, diminished vascularity, systemic diseases such as rheumatoid arthritis, gout and chronic renal failure, as well as medications including fluoroquinolones and local or systemic corticosteroids, further increase susceptibility to rupture. Case series describing asynchronous bilateral ruptures, familial clustering and associations with blood group 0 suggest that some individuals may have an inherent predisposition. Clinically, patients typically report a sudden snapping sensation or audible pop in the posterior ankle, followed by acute pain, difficulty walking and reduced plantarflexion strength. On examination, a palpable gap along the tendon, decreased resting tension and inability to perform single-leg heel rise are characteristic findings. Thompson and Matles tests play a key role in diagnosis, although partial ruptures and compensation by the plantaris and other plantar flexors may yield false-negative results. Because 20-25% of cases are initially missed, especially in middle-aged men presenting with ankle pain after sports activity, a high index of suspicion and careful clinical evaluation are essential for timely diagnosis and appropriate management.